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Acetaminophen Overdose Reveals PAR4 as a Low Expressing but Potent Receptor on the Hepatic Endothelium in Mice Arteriosclerosis, Thrombosis, and Vascular Biology Frontiers MitochondrialLysosomal Axis in Acetaminophen Hepatotoxicity Modeling and Simulation of Acetaminophen Pharmacokinetics and Hepatic Biomarkers After Overdoses of ExtendedRelease and ImmediateRelease Formulations in Healthy Adults Using the Quantitative Systems Toxicology Software Platform DILIsym Beaudoin Role of Mitochondrial Iron Uptake in Acetaminophen Hepatotoxicity In vivo upstream factors of mouse hepatotoxic mechanism with sustained hepatic glutathione depletion: Acetaminophen metabolite erythrocyte adducts and splenic macrophage generated reactive oxygen species ScienceDirect The Hidden Impact of Acetaminophen Well Rooted
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tylenol depletes glutathione pubmed Xia & He Publishing
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